If u have the hpv that causes warts does that mean ur low risk for cancer? - 3/7/2010 - HPV Warts

5 Responses to If u have the hpv that causes warts does that mean ur low risk for cancer? - 3/7/2010 - HPV Warts

1. Robin B says:

   March 7, 2010 at 7:48 am

   The HPV is a virus, weather it manifests into warts or not. The warts can be removed, but the risk for cervical cancer remains.
   References :
2. NessN says:

   March 7, 2010 at 8:27 am

   Studies indicate that women who have HPV are at a much much higher risk for cervical cancer than those who do not. Just because a strain of HPV does or doesn’t cause warts, it would still increase one’s risk of cancer.
   References :
3. fredisred08 says:

   March 7, 2010 at 8:58 am

   the real question is DO YOU HAVE HPV???
   References :
4. Corona says:

   March 7, 2010 at 9:15 am

   I did. I had one wart (gross I know). He gave me this cream that I applied to it and man it itched like crazy. I don’t know if you ever had a yeast infection, but that’s what the medicine felt like on the ONE wart. Years ago my pap turned up abnormal and then they tried to "freeze" it, and then I ended up having to have a cone biopsy, where I was told that I had pre-cancer cells. This was 6 years ago and I have been very lucky that I never had another abnormal pap, and now when I go I also request the test for hpv, which has came back negative. I guess when they did the cone biopsy they got all the bad cells. So I guess I had/have the type of hpv that causes both. Now for the first 2 years after I had it I had to go have a pap smear done every 6 months so that they could watch me closely…now I am back to my once a year visit.
   References :
5. tarnishedsilverheart says:

   March 7, 2010 at 10:04 am

   Visible genital arts are caused by low risk HPV types 6 or 11. These HPV types are rarely seen in cancer they are not HPV types that can progress to cancer. Unfortunately many of us have more than one HPV types so we can have visible genital warts which are low risk HPV types but also carry a co-infection that causes cell changes of the cervix cand could progress if not treated. It is important for women to have annual Pap test with HPV screening. A Pap test could miss abnormal cell changes.

   They are several low risk HPV types and several high risk HPV types. High risk HPV types are the types that have the potential to progress to cancer…however most high risk HPV do not progress to a cancer. Diligent screening and treatments usually prevent most HPV types from progressing.

   •The genital HPV types can be divided into two broad groups (low-risk and high-risk HPVs) depending upon their association (or lack of association) with cancers of the lower genital tract.
   •Low-risk HPV types (6, 11, 42,43,44, 54,61,70,72, and 81) are virtually never found in cancers.
   •High-risk HPV types (16, 18, 31, 33, 35, 39, 45, 51, 52, 56, 58, 59, 68, 73, and 82) have been identified in cancers of the cervix, vagina, vulva, anus, and penis.
   •The most common types detected in genital warts are HPV 6 and HPV 11.
   •The most common HPV type detected in both normal women and in women with cervical cancer is HPV 16.
   •The majority of cervical cancers (80%) are caused by just 4 HPV types (16, 18, 31, and 45).

   http://cme.asccp.org/faq/histHPV.cfm

   Genital warts
   Condylomata bearing HPV-6 or -11 have identical clinical
   manifestations and histology [2]. Recent studies have shown that about
   100% of GWs are caused by either HPV-6 or -11 but that 20–50% of
   lesions also contain co-infections with HR HPV types [3] and [4]. GWs
   do not usually result in major morbidity or mortality, but cause
   significant psychological morbidity and very substantial healthcare
   costs. Occasionally GWs persist for long periods of time and, rarely,
   such long-standing lesions may progress to malignancy. GWs are highly
   infectious, with a transmission rate of about 65% within sexual
   partnerships from an infected to a susceptible sexual partner, and an
   incubation period of between 3 weeks and 8 months, with the majority
   developing warts at around 2–3 months [3]. Once GWs have developed,
   they may show minimal change over time, become more numerous or
   larger, or regress spontaneously. The majority of placebo-controlled
   GW therapy trials show low rates of regression (around 5% complete
   clearance) in the short term, although in one study over 16 weeks 20%
   of women and 5% of men using placebo completely cleared their warts,
   and 38% of women and 22% of men using placebo cleared over 50% of
   their baseline warts [3]. Regressing warts contain significantly more
   CD4 positive T cells, both within the stroma underlying the lesions
   and the condylomata themselves, and greater expression of activation
   markers [3]. There is no report of the rate of spontaneous regression
   that may occur in the longer term. Following GW clearance with
   therapy, recurrence is common and is often seen within 3 months in 25%
   of cases, although rates of up to 67% have been observed [3]. In
   clinical practice recurrences are often seen at sites of previous
   lesions, and in these cases HPV infection in stem cells or
   slow-turnover cells at the site of previous clearance has persisted
   and then reactivated. The proportion of HPV-6/11 infections that are
   either completely cleared or persist in a latent form after clinical
   resolution is unknown, and, indeed, animal models suggest that both
   outcomes can occur [3].
   HPV-6/11 as a cause of cervical neoplasia
   HPV-6 and -11 are frequently associated with LSIL. A recent
   meta-analysis of 55 studies reported HPV-6 to be present in 8.1% of
   HPV-positive LSIL cases and HPV-11 in 3.2% of cases [25]. However, it
   remains unclear in what proportion of these HPV-6/11-positive LSIL
   cases there is concomitant co-infection with a HR type, and whether
   such HR co-infections would be "minority passenger" infections as
   described in GWs, or represent true multiple-morphology cervical lesions.
   References :